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Short Communication | 27-September-2017

Genes Controlling 2-deoxyglucose Induced Lysis and Formation of Reactive Oxygen Species in Schizosaccharomyces pombe

Schizosaccharomyces pombe cells of strains each carrying a deletion of one of the genes snf5, ypa1, pho7 and pas1 and of a strain overexpress­ing gene odr1, have been previously shown to grow in presence of the toxic glucose analogue 2-deoxyglucose (2-DG). Here we report that these genes control 2-DG induced lysis and are, with the exception of odr1, also involved in control of formation of reactive oxygen species (ROS) upon exposure of cells to H2O2. Lysis of deletion strains, but not of

Akshay Vishwanatha, Cletus J.M. D’Souza, Martin E. Schweingruber

Polish Journal of Microbiology, Volume 66 , ISSUE 3, 393–396

research-article | 30-June-2021

Effects of artemisinin and TSP-1-human endometrial-derived stem cells on a streptozocin-induced model of Alzheimer’s disease and diabetes in Wistar rats

identified in diabetes mellitus that lead to retinopathy and nephropathy (Cade, 2008). Increasing evidence demonstrates that oxidative stress has a crucial role in the expansion and progression of AD (Sharma and Gupta, 2002). Some studies proposed that amyloid-beta induces lipid peroxide, the creation of hydrogen peroxide, inflammatory cytokines, and superoxide in the brain (Esposito et al., 2006; Huang et al., 1999). Oxidative stress, through the generation of reactive oxygen species (ROS), is the main

Poorgholam Parvin, Yaghmaei Parichehreh, Noureddini Mehdi, Hajebrahimi Zahra

Acta Neurobiologiae Experimentalis, Volume 81 , ISSUE 2, 141–150

Research Article

PROBIOTICS AND THEIR POTENTIAL ANTIOXIDATIVE ACTIVITY

Nina Trojan, Paweł Satora

Postępy Mikrobiologii - Advancements of Microbiology, Volume 56 , ISSUE 1, 18–27

original-paper | 04-June-2020

N-acetylcysteine (NAC) Attenuating Apoptosis and Autophagy in RAW264.7 Cells in Response to Incubation with Mycolic Acid from Bovine Mycobacterium tuberculosis Complex

. 2018). Previous studies have shown that MTB lacking MA is less pathogenic in vivo, suggesting that MA plays an important role in MTB’s infection (Slama et al. 2016). Furthermore, MTB promotes bacterial survival by inducing oxidative stress in macrophages (Mohanty et al. 2016). Therefore, the strategy to alleviate the virulence of MTB is to inhibit the biosynthesis of MA, neutralize the oxidative stress produced by MA, and reduce the damage of ROS to intracellular substances. N-acetylcysteine (NAC

XUE LIN, MENGMENG WEI, FUYANG SONG, DI XUE, YUJIONG WANG

Polish Journal of Microbiology, Volume 69 , ISSUE 2, 223–229

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