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  • Immunohematology

 

Case report

A LU:−16 individual with antibodies

Carole Éthier, Cynthia Parent, Anne-Sophie Lemay, Nadia Baillargeon, Geneviève Laflamme, Josée Lavoie, Josée Perreault, Maryse St-Louis

Immunohematology , ISSUE 3, 110–113

Case report

Acute hemolytic transfusion reaction attributed to anti-Ata

Jay S. Raval, Sarah K. Harm, Bethann Wagner, Darrell J. Triulzi, Mark H. Yazer

Immunohematology , ISSUE 4, 140–142

Article

Anti-Vel alloimmunization and severe hemolytic disease of the fetus and newborn

risk for HDFN because transplacental passage cannot occur. Linz et al.10 recommended treatment with 2-mercaptoethanol or dithiothreitol to determine whether an IgG component is present. Both IgG and IgM had been detected in our case. A predominance of IgG in our case may well explain the severe degree of HDFN. One should assume that a paternal Vel antigen will be inherited by the fetus, given that Vel is a high-prevalence antigen. Thus, it would appear prudent to institute serial MCA-PSV

K.J. Moise, Y. Morales, M.F. Bertholf, S.N. Rossmann, Y. Bai

Immunohematology , ISSUE 4, 152–154

Case report

A suspected delayed hemolytic transfusion reaction mediated by anti-Joa

Ryan P. Jajosky, Wendy C. Lumm, Scott C. Wise, Roni J. Bollag, James F. Shikle

Immunohematology , ISSUE 2, 73–75

Review

The Augustine blood group system, 48 years in the making

The high-prevalence antigen, Ata, was first identified in 1967, but it was not until 2015 that Ata became AUG1 of a new blood group system, Augustine (AUG). The new system was established after the identification of the gene encoding Ata and the recognition of a null phenotype (AUG:–1,–2) in an At(a–) patient with an antibody (anti-AUG2) reactive with At(a–) red blood cells. The At(a–) phenotype is very rare and, with the exception of the one family with the null

Geoffrey Daniels

Immunohematology , ISSUE 3, 100–103

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