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Article | 15-February-2021

An update on the MNS blood group system

Update on the MNS Blood Group System The MNS blood group system is highly complex, with 49 antigens currently recognized by the International Society of Blood Transfusion.1 All antigens are carried by glycophorin A (GPA), glycophorin B (GPB), or multiple glycophorin (GP) variants resulting from unequal crossover or gene conversion events between GYPA and GYPB genes.2 GYPE, the other glycophorin gene family member, does not encode detectable antigens on the red blood cell (RBC) surface but has

L. Castilho

Immunohematology, Volume 35 , ISSUE 2, 61–62

Review | 20-March-2020

MNS blood group system: a review

The MNS blood group system is second only to the Rh blood group system in its complexity. Many alloantibodies to antigens in the MNS system are not generally clinically significant although antibodies to low-prevalence and high-prevalence MNS antigens have caused hemolytic disease of the fetus and newborn. The MNS antigens are carried on glycophorin A (GPA), glycophorin B (GPB), or hybrids thereof, which arise from single-nucleotide substitution, unequal crossing over, or gene conversion

Marion E. Reid

Immunohematology, Volume 25 , ISSUE 3, 95–101

Article | 14-October-2020

Low-incidence MNS antigens associated with single amino acid changes and their susceptibility to enzyme treatment

identified changes in amino acids that are associated with several low-incidence antigens in the MNS blood group system. This review relates the molecular studies with the susceptibility or resistance of these antigens to treatment of intact red blood cells by proteolytic enzymes.

Marion E. Reid, Jill Storry

Immunohematology, Volume 17 , ISSUE 3, 76–81

Review | 15-April-2020

Review: molecular basis of MNS blood group variants

chance of recombination, resulting in hybrid molecules that often carry one or more novel antigens. Some of the antigens in the MNS system result from a single nucleotide substitution. The MNS blood group system now consists of more than 40 distinct antigens. This review summarizes the molecular basis associated with some of the antigens in the MNS blood group system.

P. Palacajornsuk

Immunohematology, Volume 22 , ISSUE 4, 171–182

Article | 26-October-2020

Mta: review and case report

Anti-Mta, which recognizes an antigen in the MNS blood group system, was detected during prenatal testing of a para 6, gravida 1 woman with no history of transfusions. Her husband was apparently Mt(a–). Anti-Mta was first reported in 1962 as a naturally occurring antibody directed against a new antigen in the MNS system. The last report in the literature of detection of anti-Mta was in 1972.

Lisa Bakowski, Joanne Kosanke

Immunohematology, Volume 15 , ISSUE 2, 78–79

Article | 17-November-2020

Detection of Lewis, P1, and some MNS blood group system antibodies by a solid phase assay

Susan Rolih, Ronald Thomas, Lyle Sinor

Immunohematology, Volume 11 , ISSUE 3, 78–80

Article | 01-April-2020

An alloantibody to a highprevalence MNS antigen in a person with a GP.JL/Mk phenotype

John Ratliff, Susan Veneman, Joan Ward, Christine Lomas-Francis, Kim Hue-Roye, Randall W. Velliquette, Laima Sausais, Twilla Maldonado, Janet Miyamoto, Yolanda Martin, David Slater, Marion E. Reid

Immunohematology, Volume 23 , ISSUE 4, 146–149

Article | 21-April-2020

Severe hemolytic anemia due to auto anti-N

Caroline C. Immel, Myra McPherson, Shauna N. Hay, Linda R. Braddy, Mark E. Brecher

Immunohematology, Volume 21 , ISSUE 2, 63–65

Article | 16-February-2021

Clinical approach after identification of a rare anti-Ena in a prenatal sample

Introduction The MNS blood group system (ISBT 002) consists of red blood cell (RBC) antigens located on glycophorin A (GPA) and glycophorin B (GPB). The GYPA and GYPB genes encoding these glycophorins are located on the long arm of chromosome 4.1,2 The most frequently encountered antibodies to antigens in this system by a transfusion medicine service are those directed against M, N, S, and s. In rare cases, individuals may lack GPA or both GPA and GPB on their RBCs because of gene deletion, and

P.J. Howard, L. Guerra, D.K. Kuttner, M.R. George

Immunohematology, Volume 35 , ISSUE 4, 159–161

Article | 09-November-2020

Practical method for determination of the U status of S–s– erythrocytes

Marion E. Reid, Jill R. Storry, Joan Maurer, Sandra T. Nance

Immunohematology, Volume 13 , ISSUE 4, 111–114

Report | 25-March-2020

Molecular analyses of GYPB in African Brazilians

Ricardo Omoto, Marion E. Reid, Lilian Castilho

Immunohematology, Volume 24 , ISSUE 4, 148–153

Article | 22-January-2021

Routine indirect antiglobulin testing of blood donors—a further step toward blood safety: an experience from a tertiary care center in northern India

%] in 3 of the 19 alloimmunized donors), followed by those of the MNS blood group system (3 of 25 [12%] in 2 of 19 alloimmunized donors). Anti-K was found in one donor (1 of 25 [4%]). The majority (79%) of alloimmunized donors were D+ (15 of 19; alloimmunization rate 15 of 9779, 0.15%), and 21 percent (4 of 19; alloimmunization rate 4 of 611, 0.65%) were D– (p = 0.004; chi-square test). Of the alloantibodies to Lewis blood group system antigens, the majority were anti-Leb alone (7 of 14 donors, 50

S. Malhotra, G. Negi, D. Kaur, S.K. Meinia, A.K. Tiwari, S. Mitra

Immunohematology, Volume 36 , ISSUE 3, 93–98

Case report | 14-October-2020

Red blood cell antigen changes in malignancy: case report and review

Jeffrey L. Winters, Dianna S. Howard

Immunohematology, Volume 17 , ISSUE 1, 1–9

Report | 09-October-2019

Distribution of blood groups in the Iranian general population

%), C (2677; 77.04%), c (2557; 73.58%), and E (1059; 30.47%). In the Kell blood group system, 3293 (94.76%) samples were typed as K–k+. For the Kidd and Duffy blood group systems, the following were the most common phenotypes: Jk(a+b+) (1703; 49%), Jk(a+b–) (1006; 28.95%), Fy(a+b+) (1495; 43.02%), and Fy(a+b–) (1005; 28.92%). In the MNS blood group system, the following were the most common phenotypes: M+N+ (1668; 48%), M+N– (1310; 37.70%), S+s+ (1564; 45%), and S–s

Ehsan Shahverdi, Mostafa Moghaddam, Ali Talebian, Hassan Abolghasemi

Immunohematology, Volume 32 , ISSUE 4, 135–139

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