admission and showed a focal abnormality involving the visual cortex, calcarine region, as well as the more inferior occipital lobe and posterior temporal lobe on the right, with enhancement, FLAIR hyperintensity, but only minimal diffusion asymmetry.
On Day 7 of admission, given the lack of response to multiple antiepileptic drugs, negative lumbar puncture, and worsening MRI findings, the patient was started on methylprednisolone 1 gram IV x 3 days for presumed autoimmune encephalitis. Minimal
Svetlana P. Eckert,
North American Journal of Medicine and Science , ISSUE 1, 28–33
include leucine-rich glioma inactivated 1 (LGI1), contactin-associated protein-2 (CASPR2), and other proteins (9). The clinical characteristics of encephalitis, such as seizures, memory loss, sleep disturbance, as well as laboratory findings of hyponatremia, inflammatory cerebrospinal fluid (CSF), temporal lobe abnormalities on magnetic resonance imaging (MRI), and abnormal electroencephalography (EEG) have been previously described (10). A case of autoimmune encephalitis seropositive for NMDAR and
Scandinavian Journal of Child and Adolescent Psychiatry and Psychology , ISSUE 3, 111–115