original-paper | 08-September-2020
continuously emerged, resulting in severe problems for the clinical treatment of infectious diseases (Miyoshi-Akiyama et al. 2017).
Macrolide antibiotics not only have anti-inflammatory effects on multi-drug resistant P. aeruginosa infections (Kobayashi 1995), but also inhibit alginate production at the guanosine diphospho-d-mannose dehydrogenase (GMD) level (Mitsuya et al. 2000). Biofilm formation by P. aeruginosa is inhibited by macrolides, diminishing P. aeruginosa infection in the clinical settings. It
Polish Journal of Microbiology, Volume 69 , ISSUE 3, 349–356
Short Communication | 30-June-2018
ELIGIA M. SZEWCZYK
Polish Journal of Microbiology, Volume 67 , ISSUE 2, 237–240
original-paper | 05-December-2019
susceptibility to neomycin was interpreted according to the standards of the British Society for Antimicrobial Chemotherapy Version 14.0, 05.01.2015 (BSAC, http://bsac.org.uk/wp-content/uploads/2012/02/BSAC-Susceptibility-testing-version-14.pdf). For vancomycin, the minimal inhibitory concentration (MIC) was determined by E-test (bioMérieux, France).
The methicillin-resistant Staphylococcus aureus (MRSA) phenotype was detected using a cefoxitin disc (30 μg). The macrolide-lincosamide-streptogramin B (MLSB
Polish Journal of Microbiology, Volume 68 , ISSUE 4, 541–548