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  • Acta Neurobiologiae Experimentalis


Research paper | 15-January-2019

Calcium release from intracellular stores is involved in mitochondria depolarization after lowering extracellular pH in rat brain synaptosomes

block the metabotropic proton receptor ovarian cancer G protein-coupled recep tor 1 (OGR1). Furthermore, using fluorescent dye Fluo-3 we have demonstrated that moderate extracellular acidification induces a  cytosolic calcium increase. Excess calcium was scavenged by mitochondria (monitored by fluorescent dye Rhod-2). Our results suggest that the metabotropic OGR1 is a  hypothetical presynaptic receptor for low pH. Its activation leads to phospholipase C activation and calcium release from the

Tatsiana G. Dubouskaya, Sviatlana V. Hrynevich, Tatsiana V. Waseem, Sergei V. Fedorovich

Acta Neurobiologiae Experimentalis, Volume 78 , ISSUE 4, 343–351

Research paper | 14-August-2017

Effectors of large-conductance calcium-activated potassium channel modulate glutamate excitotoxicity
in organotypic hippocampal slice cultures

Mitochondria have been suggested as a potential target for cytoprotective strategies. It has been shown that increased K+ uptake mediate by mitochondrial ATP-regulated potassium channels (mitoKATP channel) or large-conductance Ca2+-activated potassium channels (mitoBKCa channel) may provide protection in different models of cell death. Since recent findings demonstrated the presence of BKCa channels in neuronal mitochondria, the goal of the present study was to test the potential

Marta Piwońska, Adam Szewczyk, Ulrich H. Schröder, Klaus G. Reymann, Piotr Bednarczyk

Acta Neurobiologiae Experimentalis, Volume 76 , ISSUE 1, 20–31

Research paper | 06-February-2018

Nicorandil attenuates neuronal mitochondrial dysfunction and oxidative stress associated with murine model of vascular calcification

Evidences suggest that the presence of chronic kidney disease (CKD) is associated with cerebrovascular diseases related cognitive decline in dialysis patients. As mitochondrial dysfunction is implicated in neurodegenerative disorders, we hypothesized that changes in brain mitochondria occur due to vascular calcification induced by renal failure and the opening of the mitochondrial potassium channel using nicorandil may prevent its dysfunction. Brain tissues from rats with vascular calcification

Sriram Ravindran, Krithika Swaminathan, Abhinaya Ramesh, Gino A. Kurian

Acta Neurobiologiae Experimentalis, Volume 77 , ISSUE 1, 57–67

Research paper | 10-October-2018

Effects of PINK1 mutation on synapses and behavior in the brain of Drosophila melanogaster

also detected in white mutants, which were examined to test the effect of the white background on the PINK1B9 strain. The reduced locomotor activity and longer day sleep in PINK1 mutants and after decreasing the PINK1 level in neurons seem to be correlated with a decrease in mitochondria number during the day, when they normally peak, and with impaired synaptic transmission.

Bartosz Doktór, Milena Damulewicz, Wojciech Krzeptowski, Barbara Bednarczyk, Elżbieta Pyza

Acta Neurobiologiae Experimentalis, Volume 78 , ISSUE 3, 231–241

research-article | 30-November-2018

The morphological characteristics and phylogenetic analysis of Pratylenchus vulnus Taiwan strawberry isolate

Yu-po Lin, Wan-chun Lee, Pei-che Chung, Jiue-in Yang

Journal of Nematology, Volume 51 , 1–5

Research paper | 06-February-2018

Effects of epidural compression on stellate neurons and thalamocortical afferent fibers in the rat primary somatosensory cortex

-lesion. Anterograde tracing analyses exhibited a progressive decline of TCA fiber density in barrel field for 6 months post-lesion. Due to the abrupt decrease of TCA fiber density at 3 days after compression, we further used electron microscopy to investigate the ultrastructure of TCA fibers at this time. Some TCA fiber terminal profiles with dissolved or darkened mitochondria and fewer synaptic vesicles were distorted and broken. Furthermore, the disruption of mitochondria and myelin sheath was

Tzu-Yin Yeh, Guo-Fang Tseng, Chi-Yu Tseng, Yung-Hsin Huang, Pei-Hsin Liu

Acta Neurobiologiae Experimentalis, Volume 77 , ISSUE 1, 1–17

research-article | 30-November-2018

A loop-mediated isothermal amplification assay for the plant-parasitic nematode Aphelenchoides besseyi in rice seedlings

by pestle in mortar with liquid nitrogen, and the total DNA was extracted using PowerSoil® DNA Isolation Kit (Mo Bio Laboratories Inc., CA, USA) following the manufacturer’s recommendation. Final DNA products were all stored at −20°C prior to use. LAMP primers design and reaction preparation The target gene for LAMP primers was chosen after analyzing all the available sequences of mitochondria and ribosome genes of A. besseyi and its closely related nematode species in the GeneBank database

Jiue-in Yang, Guan-yi Yu

Journal of Nematology, Volume 51 , 1–11

Review | 18-May-2018

A role for the GDAP1 gene in the molecular pathogenesis of Charcot-Marie-Tooth disease

mitochondria, where it plays a role in fission, a ubiquitous process occurring in all cells. While GDAP1 contains two glutathione S-transferase (GST) domains, its GST activity is in fact very limited. Additionally, despite GDAP1 affecting mitochondrial functionality, and hence being of great importance to cellular function, the GDAP1-associated Charcot–Marie–Tooth disease is mainly characterized by axonal degeneration. Finally, mutations in the GDAP1 gene may be inherited in a recessive or dominant manner

Weronika Rzepnikowska, Andrzej Kochański

Acta Neurobiologiae Experimentalis, Volume 78 , ISSUE 1, 1–13

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