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case-report | 31-March-2020

Infiltrative non-mass-like hepatocellular carcinoma initially presenting with isolated malignant portal vein thrombosis: A case report and review of the literature

HCC, cirrhotomimetic HCC, or cirrhosis-like HCC(6–14). Portal vein invasion is a known complicated feature of HCC, occurring by direct local venous extension or metastatic spread. However, the initial presentation of HCC as an isolated portal vein thrombosis is incredibly rare, with very few cases reported in the English literature to date(15). We, herein, present an unusual case of HCC initially presenting as an isolated malignant portal vein thrombosis without any other findings suggesting HCC

Anıl Çolaklar, Namık Kemal Altınbaş

Journal of Ultrasonography, Volume 20 , ISSUE 80, e55–e60

research-article | 30-November-2020

Prevalence of femoral vein duplication: systematic review and metaanalysis

of the femoral veins. The presence of duplicated femoral vein (DFV) is an important anatomical variation which has been shown to result in false negative compression ultrasound results during evaluation for deep venous thrombosis(2–4). The ultrasound probe may be focused on one of the branches that does not have a thrombus, whereas the thrombus may be lodged in the other branches. This may result in misdiagnosis with serious consequences such as pulmonary embolism, post-thrombotic syndrome and

William Sibuor, Vincent Kipkorir, Isaac Cheruiyot, Fidel Gwala, Beda Olabu

Journal of Ultrasonography, Volume 21 , ISSUE 87, 326–331

Case report | 31-January-2019

Portal vein tumor thrombus from gastric cancer

Sidney Ching Liang Ong, Santha Kumari Batumaly, Suryani Md Jusoh

Journal of Ultrasonography, Volume 18 , ISSUE 75, 365–368

case-report | 30-September-2019

Duplicated inferior vena cava with coexisting multiple vascular anomalies and their clinical implications: a case report

case describes duplication of the inferior vena cava coexisting with multiple anomalies of the renal and hepatic veins. Most typically, IVC anomalies are diagnosed by accident in patients who do not present any symptoms. However, such anomalies are often considered as a potential cause of deep vein thrombosis due to slow vascular flow and numerous collateral vessels. In addition, there is a risk of varicose veins due to the compression of anomalously positioned venous vessels(1). The incidence of

Cezary Gołąbek, Karolina Druć, Zuzanna Dusińska, Jakub Franke, Emil Głowacki, Agata Kuskowska, Adrianna Mróz, Oliwia Opacka, Oktawia Pięta, Anna Świderska, Karol Welc, Dominika Jaguś, Agnieszka Krauze, Maciej Jędrzejczyk

Journal of Ultrasonography, Volume 19 , ISSUE 78, 236–239

review-article | 29-January-2021


replacement therapy and oral contraceptive pill) The clinical presentation of VTE is dependent on the location of the thrombosis and may be non-specific, generally the signs and symptoms include: Deep vein thrombosis – peripheral oedema, unilateral limb pain and erythema. Pulmonary embolism – chest pain, dyspnoea, haemoptysis, tachycardia and syncope. Long-term morbidity includes pulmonary hypertension and post-thrombotic syndrome. Post-thrombotic syndrome is common and is estimated to occur in 1 in 3

Rachel MacKellar

Journal of the Australasian Society of Aerospace Medicine, Volume 12 , 25–30

Article | 15-February-2021

Serologic problems associated with administration of intravenous immune globulin (IVIg)

anemia.13,17 Thrombosis can also result from IVIg infusion, but this is quite rare compared with IVIg-associated hemolysis.18 In addition to adverse events, administration of IVIg can create challenges for the transfusion service including ABO discrepancies, positive direct antiglobulin tests (DATs), positive antibody detection tests, and incompatible crossmatches. Reverse ABO Grouping Because up to 4 g/kg of IVIg may be given to an individual with blood group A, B, or AB, these patients will often

D.R. Branch

Immunohematology, Volume 35 , ISSUE 1, 13–15

Case report | 10-January-2018

Timely Anticoagulant Thromboprophylaxis is Safe and Effective in the Care of Patients Suffering Traumatic Brain Injury.

with VTE, is responsible for implementing prevention strategies and assisting with treatment for those who unfortunately develop a Deep Vein Thrombosis or Pulmonary Embolism (PE).

Kandace Micallef

Australasian Journal of Neuroscience, Volume 25 , ISSUE 2, 30–34

Review | 31-July-2017

The physiology of blood platelets and changes of their biological activities in multiple sclerosis

Increasing evidence indicates that blood platelets contribute to diverse processes that extend beyond hemostasis. Many of the same mechanisms that play a role in hemostasis and thrombosis facilitate platelets the participation in other physiological and pathological processes, particularly in the inflammation, the immune response and central nervous system disorders. Platelets are involved in pathophysiology of central nervous system diseases, especially in the pathogenesis of multiple

Barbara Wachowicz, Agnieszka Morel, Elżbieta Miller, Joanna Saluk

Acta Neurobiologiae Experimentalis, Volume 76 , ISSUE 4, 269–281

case-report | 13-November-2017

Drain tube removal in the presence of anticoagulation in Spinal Surgery

compressive stockings, sequential compressive sleeves and the use of LMWH (standard recommendation of 40mg subcutaneously daily) all assist in the prevention of deep venous thrombosis (DVT) and pulmonary embolism (PE), the collective term being VTE (Joanna Briggs, 2016). These complications remain a major cause of morbidity and a significant cause of mortality in hospitalized patients across Australia and internationally (The Australian & New Zealand Working Party on the Management and Prevention of

Christine Holland, Sarah Smith

Australasian Journal of Neuroscience, Volume 27 , ISSUE 1, 14–18

Case report | 29-June-2016

Charcot arthropathy in ultrasound examination – a case report

autoimmune arthritis, arthritis associated with gout as well as venous thrombosis and injury. The contribution of a local inflammatory reaction and abnormal bone turnover with excessive osteoclast activity might play a role in the etiopathogenesis of this disease. As a result, osseous and articular destruction progresses rapidly leading to irreversible deformity of the foot. Avoiding weight-bearing and resting the foot in a specially selected plaster cast is the most important part of treatment. Patients

Mateusz Płaza, Anna Nowakowska-Płaza, Marta Walentowska-Janowicz, Marek Chojnowski, Iwona Sudoł-Szopińska

Journal of Ultrasonography, Volume 16 , ISSUE 65, 210–215

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